Updated HFSA MSDS provides Warning for Children

Updated Fluoridation Chemical Material Safety Data Sheet Provides Warning for Children

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July 27, 2017:

 

From Peel’s former HFSA supplier, Mosaic:

“Prolonged or repeated overexposure to fluoride compounds may cause fluorosis. Fluorosis is characterized by skeletal changes, consisting of osteosclerosis (hardening or abnormal density of bone) and osteomalacia (softening of bones) and by mottled discoloration of the enamel of teeth (if exposure occurs during enamel formation).  Symptoms may include bone and joint pain and limited range of motion. Conditions aggravated by exposure may include skin and respiratory (asthma like) disorders.”  — Pg. 3:  http://www.mosaicco.com/documents/Hydrofluosilicic_Acid_MSDS_03Jan14.pdf


“There are no children working in these chemical plants, there are no children being chemical handlers.  So this warning is for the end user.”  — Dr. Laura Pressley, PhD, Physical Chemist: https://www.youtube.com/watch?v=z1R7ami0NZQ

The Region of Peel provides no warning to residents.

Peel Public Health Staff do not monitor residents for skeletal fluorosis or attempt to properly assess for fluoride poisoning.  They dismiss dental fluorosis (hypo-mineralization, permanent harm to affected teeth) as a trivial, “merely cosmetic” effect rather than as a biomarker for fluoride toxicity.

One in 3 (34%) of the fluoridated seven year olds sampled in Dr. Dick Ito’s 2007 Peel study (funded by the Region) had dental fluorosis.  Four percent of Peel children had moderate or severe dental fluorosis according to the Region’s 2003 oral health report.

Earlier this year, Peel Public Health, desperate to cover-up the extent of harm, published an unscientific (and in my mind, obviously fraudulent) report claiming that miraculously only 2.1% of Peel students with a dental assessment are now affected by dental fluorosis.  No one has been willing to provide any meaningful details on this statistic, which is apparently based on children too young to properly assess for dental fluorosis.


[The screen shots below are from FFP’s Feb. 2017 delegation to Council: https://www.fluoridefreepeel.ca/wp-content/uploads/2017/02/FFP-delegate-Feb-9-2017-v2-1-slide-per-page.pdf]

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National Research Council. 2006. Fluoride in Drinking Water: A Scientific Review of EPA’s Standards. Washington, DC: The National Academies Press.:

ENAMEL FLUOROSIS

Fluoride has a great affinity for the developing enamel because tooth apatite crystals have the capacity to bind and integrate fluoride ion into the crystal lattice (Robinson et al. 1996). Excessive intake of fluoride during enamel development can lead to enamel fluorosis, a condition of the dental hard tissues in which the enamel covering of the teeth fails to crystallize properly, leading to defects that range from barely discernable markings to brown stains and surface pitting. This section provides an overview of the clinical and histopathological manifestations of enamel fluorosis, diagnostic issues, indexes used to characterize the condition, and possible mechanisms.

Clinical and Histological Features

Enamel fluorosis is a mottling of the tooth surface that is attributed to fluoride exposure during tooth formation. The process of enamel maturation consists of an increase in mineralization within the developing tooth and concurrent loss of early-secreted matrix proteins. Exposure to fluoride during maturation causes a dose-related disruption of enamel mineralization resulting in widening gaps in its crystalline structure, excessive retention of enamel proteins, and increased porosity. These effects are thought to be due to fluoride’s effect on the breakdown rates of matrix proteins and on the rate at which the by-products from that degradation are withdrawn from the maturing enamel (Aoba and Fejerskov 2002).

Clinically, mild forms of enamel fluorosis are evidenced by white horizontal striations on the tooth surface or opaque patches, usually located on the incisal edges of anterior teeth or cusp tips of posterior teeth. Opaque areas are visible in tangential reflected light but not in normal light. These lesions appear histopathologically as hypomineralization of the subsurface covered by a well-mineralized outer enamel surface (Thylstrup and Fejerskov 1978). In mild fluorosis, the enamel is usually smooth to the point of an explorer, but not in moderate and severe cases of the condition (Newbrun 1986). In moderate to severe forms of fluorosis, porosity increases and lesions extend toward the inner enamel. After the tooth erupts, its porous areas may flake off, leaving enamel defects where debris and bacteria can be trapped. The opaque areas can become stained yellow to brown, with more severe structural damage possible, primarily in the form of pitting of the tooth surface.

Enamel in the transitional or early maturation stage of development is the most susceptible to fluorosis (DenBesten and Thariani 1992). For most children, the first 6 to 8 years of life appear to be the critical period of risk.  https://www.nap.edu/read/11571/chapter/6#104

The Region is causing permanent harm to teeth, in the name of oral health And this is just the tip of the iceburg of harm.  For example:

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Not surprisingly, it looks like Mosaic would prefer we not find their HFSA MSDS that includes a WARNING FOR CHILDREN.

Mosaic no MSDS